- Following 7K
- Followers 239.6K
- Engagement 0.88%
- Avg likes 2.1K
- Avg comments 111
About MOVABILITY
MOVABILITY (@movability) is a Health & wellness website creator on Instagram. The account has 239,623 followers and 890 published posts. Recent posts average 2,104 likes and 111 comments, an engagement rate of 0.88%.
An audience of 239.6K puts MOVABILITY in the mid-tier, between 100,000 and 1 million followers. Set against 7,028 accounts followed, the audience works out to about 34 followers per account MOVABILITY follows. MOVABILITY is set up as an Instagram business account. Flinque files MOVABILITY under a single category, Health & wellness website.
The Instagram bio for MOVABILITY runs to 24 words across 4 lines. The bio tags #movability. The latest activity Flinque has on record for MOVABILITY dates from December 2025.
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890 Posts
Of the 10 most recent posts shown for MOVABILITY, 9 are videos and 1 is a single image. That mix leans toward videos. MOVABILITY has built a substantial archive of 890 posts. Against the audience, that is roughly 269 followers for each post published. MOVABILITY also keeps 11 story highlights on the profile. All 10 carry captions, averaging 316 words each. The captions are written mainly in English. Words that recur across them include sina, often, itâs, brain and symptoms.
Anabolic resistance = a blunted muscle protein synthesis response to resistance training and dietary protein. Same meal, same workout, smaller return. Under the hood: amino acids, leucine, mTOR, insulin signaling.
Big drivers I see:
⢠Aging: the âprotein thresholdâ per meal rises with age.
⢠Menopause: many women notice a sharper drop in strength and recovery post-menopause.
⢠Inactivity: low steps, injury, bed rest, travel, even a couple weeks of deconditioning can switch muscle into âdonât invest hereâ mode.
⢠Metabolic stress: obesity, insulin resistance, chronic low-grade inflammation.
⢠Post-viral states: influenza, COVID-19, mono/EBV and other latent viruses can leave behind inflammation, appetite loss, mitochondrial dysfunction, and prolonged weakness (long COVID and post-viral fatigue are common). Cytokines like IL-6 and TNF-ι can push breakdown up and block the normal anabolic signal.
What helps, consistently:
1. Lift 2-4x/week with progressive overload.
2. Keep daily activity high. Steps matter.
3. Hit protein daily and per-meal, and distribute it (donât skip breakfast protein).
⢠Training: ~1.4-2.0 g/kg/day
⢠Older or rebuilding: ~1.2-1.6 g/kg/day
⢠Per meal: younger ~0.25 g/kg, older closer to ~0.4 g/kg (often 25-40 g/meal)
4. Prioritize leucine-rich, complete proteins (whey, dairy, eggs, meat, fish, soy). Plant-based works, but you may need more total protein and smarter combos.
5. Supportive add-ons: creatine (3-5 g/day), omega-3s, vitamin D if deficient, pre-sleep protein if totals are hard.
6. Donât ignore sleep and circadian rhythm, and gut health. Both can shift inflammation and insulin sensitivity.
This is something I screen for in a patient history because it changes the whole plan. Educational info only, work with your clinician.
For a deep dive, subscribe to my Substack: Movability masterclass. At Movability we take a collaborative approach, naturopathic testing when appropriate, rehab programming, and coordination with your doctor.
â Dr. Sina
Sciatica is a symptom pattern (lumbar radicular pain): a low back nerve root gets irritated and you feel pain, tingling, numbness, or weakness travel into the leg. The most common driver is disc pathology (a bulge or herniation). In older bodies, stenosis and bony overgrowth become more common contributors.
If your âsciaticaâ doesnât behave like nerve-root pain, investigate common look-alikes. I think in 3 buckets:
1. Referred pain generators (not a pinched nerve)
⢠SI joint dysfunction or sacroiliitis
⢠Facet joint referral (often worse with extension/rotation)
⢠Hip joint pathology (arthritis, labral issues, groin/anterior thigh pain)
⢠Greater trochanteric pain syndrome (bursitis/glute tendons, hurts to lie on it)
⢠Proximal hamstring tendinopathy or ischial bursitis (âsit boneâ pain)
2. Peripheral nerve and extra-spinal entrapments
⢠Deep gluteal syndrome/piriformis region irritation
⢠Superior cluneal nerve irritation (iliac crest pain that can refer down)
⢠Meralgia paresthetica (burning outer thigh)
⢠Common peroneal neuropathy (top of foot numbness, possible foot drop)
3. System and vascular drivers
⢠Peripheral neuropathy (often bilateral burning/numbness in the feet)
⢠Vascular claudication/PAD (cramping with walking, better with rest, check pulses)
⢠Inflammatory back pain patterns (morning stiffness, better with movement)
Three questions that clarify fast: where does it start, how far does it travel, and do you have numbness, tingling, or weakness?
Red flags: new bowel/bladder changes, saddle numbness, fever, unexplained weight loss, or rapidly progressive weakness.
The full Root Cause Breakdown is already live in Movability Masterclass on Substack, link at the top of my page.
â Dr. Sina
Functional Neurological Disorder (FND) is a real brain based condition where movement, sensation, speech, gait, or seizure like episodes malfunction without structural damage on routine scans. Itâs not âfaking,â and itâs not âjust anxiety.â In neurology clinics itâs a top reason for referral (often 5 to 10% of new visits).
What research supports:
⢠Network disruption linking emotion (amygdala), salience/interoception (insula, cingulate), and sensorimotor control.
⢠Predictive processing gets stuck, expectations override real time signals, symptoms become involuntary and âlearned.â
⢠Impaired sense of agency, the brain mislabels actions as not self generated.
⢠Autonomic imbalance is common (high arousal, lower vagal tone/HRV), which can fuel dizziness, tremor, shutdown, and faintness.
⢠Brainstem involvement, altered limbic to brainstem pathways tied to threat and autonomic regulation.
Now the piece most people skip: the upper neck.
Upper cervical proprioceptors feed the brainstem. After whiplash, hypermobility, chronic neck tension, or vestibular issues, that input can become noisy. In some people it perpetuates FND patterns, and in others it mimics them, so it must be assessed.
Misdiagnosis and overlap matter. FND can be mistaken for, or coexist with, epilepsy, stroke/TIA, MS, vestibular migraine/BPPV, and POTS or other dysautonomia. A solid FND diagnosis is based on positive clinical signs (like Hooverâs sign, tremor entrainment), not just âtests are normal.â
Who gets it more? In adult clinics, women are diagnosed more often (often around 60 to 80%), likely due to a mix of biology, stress load, trauma exposure, and bias. Men and kids can get it too.
Iâm a chiropractor and global complex case consultant. I support complex cases by addressing brainstem inputs, neck proprioception, vestibular and visual coordination, breathing, autonomic regulation, and movement retraining, alongside neurology, PT/OT, and therapy when needed.
Want the deep dive? My Movability Masterclass on Substack breaks it down. Link at the top of my page.
â Dr. Sina
In one post-concussion athlete survey, 54% rated loss of or poor appetite as one of their most severe symptoms.
Hereâs why: concussions can disrupt your hormone system, the invisible web that controls stress, blood sugar, metabolism, and hunger. Even mild TBIs can affect the hypothalamus and pituitary, the brainâs hormone control center. Cortisol (your stress response hormone) can be dysregulated, too high, too low, or mistimed, and that can blunt appetite and destabilize energy. Insulin sensitivity can also shift after TBI, and insulin resistance has been documented in moderate-to-severe cases. Thyroid hormones can dip after head injury and illness stress, potentially slowing digestion and dulling hunger. Even appetite signaling can change: hormones like ghrelin (hunger) and leptin (satiety) may get out of sync too. Across moderate-to-severe TBI, meta-analyses suggest pituitary hormone deficits in about 1 in 4. Even after mild TBI, a 2024 study of female athletes found pituitary dysfunction in 12.2%.
If this is happening to you, youâre far from alone. Yet appetite and hormone changes are often overlooked in concussion recovery. You might have been told your lingering symptoms were just anxiety, or that appetite loss isnât related to your injury. That dismissal is brutal when your physiology is clearly dysregulated.
At Movability, we take this into account. When the pattern fits, we run targeted labs through our naturopath and coordinate with your physician so care stays aligned.
Deep dive this topic with me on MOVABILITY MASTERCLASS on Substack, link at the top of my page.
â Dr. Sina
Whatâs happening: the smell system lives in a small patch high in the nasal cavity (olfactory epithelium). Viruses can inflame this area and disrupt the support cells that keep smell neurons working. When support cells are damaged, smell neurons lose their normal environment, odor-receptor genes get turned down, and signaling to the brain drops. Thatâs why you can lose smell even without congestion, and why recovery can be slow. Itâs tissue repair plus nerve regeneration plus brain rewiring.
Typical timeline (non-linear):
⢠1â4 weeks: many get partial return.
⢠4â12 weeks: progress can be stop-and-start. Parosmia often appears during regrowth.
⢠3+ months: still recoverable, but consider an ENT evaluation, especially if itâs one-sided, worsening, or paired with severe headaches, vision changes, or new neurologic symptoms.
How to get your sense of smell back at home (the basics that show up across high-quality research):
1. Olfactory training (smell training) 2x/day for 12 weeks: rose, lemon, clove, eucalyptus. 20 seconds each. Gentle sniffs. Focus on the memory of the scent.
2. Nasal care: saline spray or rinse to keep the lining healthy (distilled or boiled-then-cooled water).
3. Reduce inflammation: manage allergies, sleep, protein, hydration. Omega-3s are a reasonable low-risk support.
4. Track progress weekly with a simple sniff test (coffee, citrus peel, vanilla, soap). Look for trends over weeks, not day-to-day.
5. Avoid zinc nasal sprays and harsh âburningâ remedies. These can permanently damage smell.
6. If âtasteâ feels gone, itâs often smell. Lean on texture, temperature, acid, and spice while you recover. Add smoke and gas detectors for safety.
Want the full deep dive, plus the exact protocol, troubleshooting for parosmia, and a printable routine? Movability Masterclass on Substack, link at the top of my page.
â Dr. Sina
Iâm Dr. Sina Yeganeh, D.C., co-founder of Movability, and I work with complex chronic cases alongside healthcare professionals. One of the most overlooked patterns I see is the gastro-vestibular, autonomic link: GERD or silent reflux (LPR, laryngopharyngeal reflux) showing up as vertigo, lightheadedness, rocking, or brain fog.
Hereâs the physiology map most people miss:
1. Vagus nerve reflexes. Acid and inflammation in the esophagus can fire vagal afferents into the brainstem and shift autonomic output and baroreflex tone. Translation: palpitations, air hunger, blood pressure drops, and that âI might faintâ dizziness.
2. The ear route (Eustachian tube dysfunction). LPR can inflame the nasopharynx and Eustachian tubes, altering middle ear pressure and irritating the balance system. That can look like true vertigo, ear fullness, tinnitus, popping, and âboatâ imbalance.
3. Mechanics and pressure. Hiatal hernia, constipation, bloating, bracing, bending forward, and sleeping flat can increase reflux events. Sleep position matters for many people, right side and flat often worsen it, left side and head elevation often help.
4. The constellation. hEDS, POTS (dysautonomia), MCAS, histamine intolerance, food triggers, and vestibular migraine can stack, one trigger can drive reflux and dizziness at the same time.
5. Plot twist: treatment. PPIs, H2 blockers (famotidine), and prokinetics (metoclopramide) can cause dizziness in some people. Timing matters.
Not medical advice. If you have fainting, chest pain, new neurologic symptoms, or severe worsening, get evaluated. Otherwise, track patterns: meals, posture, sleep side, neck position, meds, triggers.
Full Root Cause Breakdown, including how we approach this collaboratively inside Movability and a real-world case vignette, is on my Substack: Movability Masterclass.
Pain is an output of your nervous system, not a perfect tissue damage meter. Itâs built from sensory input plus context, stress, sleep, past injury, and perceived threat. When your brain decides âdanger,â it turns up the volume on pain, increases muscle tone, and narrows attention onto the sore spot. That attention lock is not weakness, itâs a survival circuit (salience network, insula, ACC).
This exercise is basically somatic tracking with a safety anchor (pain reprocessing principles). You train attention to move between âthreatâ and âsafety.â When you anchor in a safe body sensation, you can support parasympathetic regulation and tap into descending pain inhibition, your built-in braking system.
RED + BLUE CIRCLE RESET (2 to 4 minutes)
1) Lie down supported. Eyes closed. Slow your exhale.
2) RED circle: choose ONE painful or tight spot. Mentally draw a red circle around it. Observe for 10 to 15 seconds. Label sensations (pressure, pulling, heat, buzzing). No fixing.
3) BLUE circle: choose ONE area that feels neutral or good (hand warmth, belly breath, feet, jaw). Draw a blue circle. Stay 20 to 30 seconds. Longer exhale. Soften your face.
4) Alternate 3 to 6 rounds. Spend more time in BLUE than RED. Always finish in BLUE.
Who this helps most:
⢠chronic tightness and guarding
⢠pain that spikes with stress or anxiety
⢠persistent pain, central sensitization patterns
⢠post-injury fear of movement, flare-ups that feel âbiggerâ than the tissue story
Real talk, this is not the only answer. Chronic pain usually needs a toolkit, education, graded exposure, strength, sleep, stress support, and systems based care. But for many people, learning âI can notice pain AND access safetyâ is a turning point.
Pro tip, if you canât find a âgoodâ spot, use neutral. Feel calves on the table, air at your nostrils, or tongue resting. This is interoception training. Do it 1 to 3 times a day, before stretching or sleep.
â Dr. Sina
One small muscle under the jaw can blur the lines between dentistry, ENT, neurology, and musculoskeletal care: the digastric.
This is a pattern I screen for constantly in complex chronic pain. In the clip, Iâm palpating under the mandible while my patient swallows and gently opens her jaw. That visible contraction matters, especially when it reproduces a familiar symptom.
Why it matters clinically:
The digastric has two parts and two different cranial nerve control systems. The front portion is driven by trigeminal pathways (CN V3), the back portion by facial nerve pathways (CN VII). It influences jaw opening, hyoid positioning, swallow mechanics, and tongue base coordination. When itâs irritated, traumatized, or stuck in a protective bracing program, it can refer symptoms into the lower front teeth, the throat, and the ear region, while also feeding clenching, TMJ overload, and forward head posture.
And in certain anatomies, this area sits in a crowded neighborhood next to major vessels and cranial nerves. If trauma, guarding, or scar tissue increases mechanical crowding, it may contribute to a pressure type symptom picture, pulsatile whooshing tinnitus, dizziness, or brain fog that behaves mechanically.
Iâm Dr. Sina Yeganeh, DC, co-founder of Movability, and a global complex case consultant. Clinicians, add a 30 second swallow and suprahyoid screen. Patients, advocate for a full system map.
Key clues I listen for: symptoms fluctuate with chewing gum, long conversations, singing, swallowing saliva, or neck rotation and extension. Many people get reassured by normal scans, but function problems can still be present. This is why I teach pattern recognition, not protocols.
I broke down the full reasoning and a real case analysis in Movability Masterclass on Substack. Link in bio.
Thank you for helping me demo
â Dr. Sina
Let me be very clear: normal imaging doesnât mean youâre fine. It means theyâre not looking in the right place.
Patients come in with numbness in the ring and pinky fingers, snapping at the elbow, and weakness when gripping. Theyâre told itâs nothing ⤠because imaging and nerve tests are normal. But the symptoms arenât.
What they often have is a mechanical entrapment of the ulnar nerve đ not at the elbow where most people look, but proximal to it, where the nerve pierces the medial intermuscular septum and runs along the medial head of the triceps. That fascial tension pulls on the cubital tunnel, making the nerve snap over the medial epicondyle with flexion.
By the time they get to me, theyâve already tried bracing, rest, stretching, maybe even physio. But none of that works if the nerve canât glide.
This is what happens when we reduce nerve pain to âpinched nerves.â We miss the tension đ the loss of nerve excursion đ the altered neurodynamics đ concepts well-documented in the literature but rarely applied in practice.
In this video, Iâm using my Movability Neuromuscular Release Technique to trace and release the full length of the ulnar nerve âĄď¸from the neck to the handâĄď¸clearing tension at every point it can get stuck: brachial fascia, medial septum, triceps, biceps aponeurosis, cubital tunnel, FCU, and Guyonâs canal.
Nerves donât just get compressed đŁ they get tethered, starved, and irritated. Even a 6â8% stretch can reduce blood flow. Over time, tension disrupts axoplasmic flow, triggers inflammation, and builds hypersensitivity in the nerve itself. If it canât glide, it wonât heal. Thatâs why we treat the whole pathânot just where it hurts.
Itâs not magic đŞ itâs anatomy. You just have to know where to look.
When you restore glide, you restore function.
â Dr. Sina
Your neck is more than just a support for your headâitâs a powerhouse of sensors and blood flow pathways that play a key role in balance. When something goes wrong in the cervical spine, it can throw off your bodyâs ability to keep you steady. Hereâs why:
đĄ Proprioception:
The upper cervical spine (especially C1-C3) is packed with tiny sensors that tell your brain where your head is in space. If these sensors send mixed signalsâdue to stiffness, poor posture, or injuryâit creates confusion in your brainâs balance centers, making you feel unsteady.
đĄ Blood Flow:
The vertebral arteries run through your neck, supplying blood to the brainstem and inner ear. If neck dysfunction compresses or irritates these arteries, your brain may not get the blood flow it needs, leading to dizziness.
đĄ Muscle Tension:
Tight muscles like the suboccipitals or SCM (sternocleidomastoid) can affect both blood flow and the feedback your brain gets about movement. This tension amplifies the problem, leaving you feeling off balance.
How can you tell the difference between cervicogenic dizziness and vertigo?
Cervicogenic dizziness feels more like unsteadiness or a floating sensation, often linked to neck pain, stiffness, or posture. Itâs closely tied to neck movementâturning your head or holding your neck in a certain position can trigger symptoms.
In contrast, vertigo causes a spinning sensation, often from inner ear issues, and usually isnât related to neck movement.
The good news? Cervicogenic dizziness is treatable! By addressing the root causesâwhether itâs joint dysfunction, posture, or muscle tensionâwe can help restore normal function and bring back your balance. đ
If youâve been struggling with dizziness, itâs time to stop guessing and start addressing the real issue. Drop your questions below or book an assessment today to get to the root cause!
MOVABILITY's engagement
MOVABILITY's engagement rate on Instagram is 0.88%, under 1%, fewer than 1 like or comment per 100 followers. On a typical post that comes to about 1 like or comment for every 114 followers. An average post collects 2,104 likes and 111 comments, close to 19 likes for each comment.
Engagement rate
0.88%
- Avg likes
- 2.1K
- Avg comments
- 111
- Interactions : followers
- 1 : 114
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Who is MOVABILITY?
MOVABILITY (@movability) is a Health & wellness website creator on Instagram. MOVABILITY is set up as an Instagram business account. The Instagram bio for MOVABILITY runs to 24 words across 4 lines.
How many followers does MOVABILITY have?
MOVABILITY has 239,623 followers on Instagram (239.6K). Set against 7,028 accounts followed, the audience works out to about 34 followers per account MOVABILITY follows. MOVABILITY has built a substantial archive of 890 posts.
What is MOVABILITY's engagement rate?
MOVABILITY's engagement rate on Instagram is 0.88%, under 1%, fewer than 1 like or comment per 100 followers. On a typical post that comes to about 1 like or comment for every 114 followers. An average post collects 2,104 likes and 111 comments, close to 19 likes for each comment.
What does MOVABILITY post about on Instagram?
Flinque files MOVABILITY under a single category, Health & wellness website. Of the 10 most recent posts shown for MOVABILITY, 9 are videos and 1 is a single image. The 10 recent Instagram captions Flinque holds for MOVABILITY repeatedly use the words sina, often, itâs, brain and symptoms. MOVABILITY writes those captions mainly in English. The bio tags #movability. MOVABILITY keeps 11 story highlights, which open with a free Flinque account.
How do I contact MOVABILITY for a collaboration?
MOVABILITY's contact details are not published on Flinque's public profile. The Instagram bio links to 1 external site, and a free Flinque account opens that link. The account is registered as an Instagram business account. Brands with a free Flinque account can shortlist MOVABILITY and use Flinque's outreach tools wherever a contact route is on file.
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